Custom Models
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- ● Diet-Induced Obesity (DIO) Mouse Model
- ● Type 1 Diabetes Mellitus (T1DM) Mouse Model
- ● Type 2 Diabetes Mellitus (T2DM) Mouse Model
- ● Diet-induced Metabolic Dysfunction-Associated Steatotic Liver Disease (MASLD) Mouse Model
- ● Chemically Induced Metabolic Dysfunction-Associated Steatotic Liver Disease (MASLD) Mouse Model
- ● Metabolic Dysfunction-Associated Steatotic Liver Disease (MASLD) Mouse Model (Gene-editing)
- ● Composite (Combined) Model - Metabolic Dysfunction-Associated Steatotic Liver Disease (MASLD) Model
- ● Composite Model - Atherosclerosis Mouse Model
- ● Atherosclerosis Mouse Model (Gene-editing)
- ● Acute Pancreatitis Mouse Model
- ● Chronic Pancreatitis Mouse Model
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Testimonials
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Citations
IMMUNITY 47:107 (2017) IF=22.845
B6J-hC5 Mice
Figure 1. Fundus morphology and OCT results of C57BL/6JCya and B6J-hC5 mice
The fundus morphology and retinal OCT results of heterozygous and homozygous B6J-hC5 mice were consistent with those of C57BL/6JCya.
Figure 2. Electroretinogram (ERG) detection results of C57BL/6JCya and B6J-hC5 mice
Compared with C57BL/6JCya, the amplitudes of the a-wave and b-wave in both scotopic and photopic ERG recordings of heterozygous and homozygous B6J-hC5 mice were nearly identical to those of the C57BL/6JCya. The retinal photoreceptor function of heterozygous and homozygous B6J-hC5 mice were normal.
